Team: Stanford (California)
Journal of Translational Medicine
Wnt protein by activating the stem cells of the bone is the origin of the resconstruction bone after a fracture, for example. Researchers have succeeded in improving the speed of fracture healing in mice genetically modifying them to over-activate the Wnt pathway.
Moreover, by administering the protein Wnt3 in liposomes in normal mice (vectorization), they were able to increase the speed of their bone repair without generating excessive formation of bone that was feared. This study opens
clinical pathways in the repair of fractures, but also of stroke, heart attacks or skin lesions.
The Wnt protein pathway (related to cadherin)
http://vulgariz.com/medecine-sante/cellules-souches/accelerer-la-reparation-dune-fracture-osseuse/
0 comments:
Post a Comment